@misc{10481/91535,
year = {2016},
month = {12},
url = {https://hdl.handle.net/10481/91535},
abstract = {Over recent years, it has been proposed that some diseases of unknown origin, such as 
schizophrenia, may be caused by persistent chronic infections coupled with a genetic 
component and may be perpetuated by the immune system. This hypothesis is supported 
by epidemiological and biological evidence on the exposure of schizophrenics to infec‐
tious diseases during prenatal or postnatal periods, including Toxoplasma gondii, chla‐
mydia, human herpes virus, human endogenous retroviruses, parvovirus B19, mumps, 
and flu viruses. This growing list of microbes will undoubtedly continue to increase in 
the future. Linking infection to schizophrenia is a complex challenge that requires further 
experimental and epidemiological research. T. gondii is the infectious agent that has most 
frequently been related to neuropsychiatric disorders, including schizophrenia, and it is 
considered to represent a highly useful model to analyze the influence of a microorgan‐
ism on human behavior and the development of psychiatric disease. It may also help 
to detect patient subpopulations susceptible to treatment with specific antimicrobials 
by improving definition of the differential phenotype of the disease, and it offers the 
possibility of a preventive approach.},
publisher = {IntechOpen},
keywords = {Schizophrenia},
keywords = {Toxoplasma gondii},
keywords = {Antibodies},
keywords = {Behavior},
keywords = {Cytokine},
keywords = {Neurotransmitter},
keywords = {Gene‐infection interaction},
title = {Toxoplasma gondii and Schizophrenia: A Relationship That Is Not Ruled Out},
doi = {10.5772/66018},
author = {Sorlozano Puerto, Antonio and Gutiérrez Fernández, José},
}